GLP-1 / GIP (T)
GLP-1 / GIP (T)
Weight Management & Metabolism
GLP-1/GIP (Tirzepatide) is a dual-action peptide targets both GLP-1 and GIP receptors, two key pathways involved in metabolic regulation. It is widely studied for its potential to enhance weight management, regulate blood sugar levels, and improve overall metabolic efficiency.
- ✓ ≥99% purity
- ✓ Third-party COA
- ✓ US-synthesized
- ✓ Lyophilized powder
GLP-1 Tirzepatide is studied to better understand how two separate metabolic receptor pathways work together to influence insulin response, blood sugar regulation, and energy metabolism.
Tirzepatide belongs to a new class of dual incretin receptor agonists, built on the observation that GIP and GLP-1 — the two primary incretin hormones released by the intestine in response to nutrient intake — operate through complementary but distinct signaling pathways. Engaging both simultaneously in laboratory models produces measurably different downstream outcomes compared to either pathway alone, making Tirzepatide a uniquely informative tool compound for metabolic signaling research.
Tirzepatide also demonstrates the ability to increase adiponectin levels in adipocyte cell preparations — an adipokine associated with lipid metabolism regulation and energy expenditure signaling — adding a third dimension to its research utility beyond pancreatic incretin biology.
- CAS Number: 2023788-19-2
- Molecular Weight: 4813.5 g/mol
- Purity: ≥99%
- Also Known As: Tirzepatide, GLP-1/GIP dual agonist, LY3298176 (development code), Mounjaro (pharmaceutical formulation — not supplied here)
- Chemical Formula: C₂₂₅H₃₄₈N₄₈O₆₈
In cell-based and preclinical models, Tirzepatide engages GIPR and GLP-1R through distinct but complementary mechanisms. At GIPR, it mimics native GIP with comparable affinity, activating Gs-coupled adenylyl cyclase signaling and intracellular cAMP accumulation in beta-cell preparations. At GLP-1R, its biased agonism preferentially drives cAMP synthesis over beta-arrestin recruitment — a signaling preference believed to underlie the enhanced insulinotropic response observed in dual-receptor activation models relative to GLP-1 monotherapy preparations.
The combined GIPR and GLP-1R activation produces a synergistic effect on insulin secretion signaling and glucagonostatic activity in pancreatic islet preparations, exceeding the responses generated by either receptor pathway in isolation. This synergy is the primary pharmacological feature studied in incretin biology research using Tirzepatide as a tool compound.
- Dual Incretin Receptor Biology
- Biased Agonism Research
- Insulin Secretion Pathway Studies
- Glucagonostatic Signaling Research
- Adipokine and Lipid Metabolism Studies
- Incretin Synergy Modeling
- Metabolic Signaling Research
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